Open Access

Erratum to: dysregulation of matricellular proteins is an early signature of pathology in laminindeficient muscular dystrophy

  • Thomas Mehuron1Email author,
  • Ajay Kumar1,
  • Lina Duarte1,
  • Jenny Yamauchi1,
  • Anthony Accorsi1 and
  • Mahasweta Girgenrath1
Skeletal Muscle20144:17

DOI: 10.1186/2044-5040-4-17

Received: 18 September 2014

Accepted: 18 September 2014

Published: 2 October 2014

The original article was published in Skeletal Muscle 2014 4:14

Correction

After publication of this work [1], we noted that while we were initially going to include muscle function tests in this study, as our manuscript evolved and we decided to include earlier time points for characterization, we decided to remove it because pups younger than 2 weeks cannot be measured. While we removed this from the results, we missed retracting them from the methods section of the abstract. Therefore, the methods section of the abstract should instead read:

Methods

We sought out to examine the dysregulation of various pathways throughout early development (postnatal weeks 1–4) in the DyW mouse, the most commonly used mouse model of laminin-deficient muscular dystrophy. Gene (qRT-PCR) and protein levels (western blot, ELISA) as well as histology (H&E, picrosirius red staining) and immunohistochemistry (fibronectin, TUNEL assay) were used to assess dysregulation of matricellular proteins.

Notes

Authors’ Affiliations

(1)
Department of Health Sciences, Boston University

References

  1. Mehuron T, Kumar A, Duarte L, Yamauchi J, Accorsi A, Girgenrath M: Dysregulation of matricellular proteins is an early signature of pathology in laminin-deficient muscular dystrophy. Skeletal Muscle 2014, 4: 14. 10.1186/2044-5040-4-14PubMed CentralView ArticlePubMedGoogle Scholar

Copyright

© Mehuron et al.; licensee BioMed Central Ltd. 2014

This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.

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