Fig. 9From: VAChT overexpression increases acetylcholine at the synaptic cleft and accelerates aging of neuromuscular junctionsAnalysis of transcripts associated with muscle fiber identity (MyHC2A, MyHC2B, MyHC2B) and markers of muscle regeneration and atrophy (Myogenin, Foxo1, Atrogin-1) revealed no change in the EDL muscle of VAChTHyp compared to control mice at 5 and 19 months of age (a–f). At least four female mice per genotype maintained on a C57BL/6 background were used to compare transcript levels. Error bar = Standard ErrorBack to article page