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  1. The repair process of damaged tissue involves the coordinated activities of several cell types in response to local and systemic signals. Following acute tissue injury, infiltrating inflammatory cells and resi...

    Authors: Christopher J Mann, Eusebio Perdiguero, Yacine Kharraz, Susana Aguilar, Patrizia Pessina, Antonio L Serrano and Pura Muñoz-Cánoves
    Citation: Skeletal Muscle 2011 1:21
  2. During development, the branchial mesoderm of Torpedo californica transdifferentiates into an electric organ capable of generating high voltage discharges to stun fish. The organ contains a high density of cholin...

    Authors: Suzanne E. Mate, Kristy J. Brown and Eric P. Hoffman
    Citation: Skeletal Muscle 2011 1:20
  3. Excitation-contraction coupling involves the faithful conversion of electrical stimuli to mechanical shortening in striated muscle cells, enabled by the ubiquitous second messenger, calcium. Crucial to this pr...

    Authors: EMichelle Capes, Randall Loaiza and Héctor H Valdivia
    Citation: Skeletal Muscle 2011 1:18
  4. Leukemia inhibitory factor (LIF) is known to inhibit myogenic differentiation as well as to inhibit apoptosis and caspase-3 activation in non-differentiating myoblasts. In addition caspase-3 activity is requir...

    Authors: Liam C Hunt, Aradhana Upadhyay, Jalal A Jazayeri, Elizabeth M Tudor and Jason D White
    Citation: Skeletal Muscle 2011 1:17
  5. Stromal interaction molecules (STIM) were identified as the endoplasmic-reticulum (ER) Ca2+ sensor controlling store-operated Ca2+ entry (SOCE) and Ca2+-release-activated Ca2+ (CRAC) channels in non-excitable cel...

    Authors: Santeri Kiviluoto, Jean-Paul Decuypere, Humbert De Smedt, Ludwig Missiaen, Jan B. Parys and Geert Bultynck
    Citation: Skeletal Muscle 2011 1:16
  6. Oculopharyngeal muscular dystrophy (OPMD) is a late-onset progressive muscle disorder caused by a poly-alanine expansion mutation in the Poly(A) Binding Protein Nuclear 1 (PABPN1). The molecular mechanisms tha...

    Authors: Seyed Yahya Anvar, Peter A. C. 't Hoen, Andrea Venema, Barbara van der Sluijs, Baziel van Engelen, Marc Snoeck, John Vissing, Capucine Trollet, George Dickson, Aymeric Chartier, Martine Simonelig, Gert-Jan B. van Ommen, Silvere M. van der Maarel and Vered Raz
    Citation: Skeletal Muscle 2011 1:15
  7. A hallmark of muscular dystrophies is the replacement of muscle by connective tissue. Muscle biopsies from patients severely affected with facioscapulohumeral muscular dystrophy (FSHD) may contain few myogenic...

    Authors: Guido Stadler, Jennifer CJ Chen, Kathryn Wagner, Jerome D Robin, Jerry W Shay, Charles P Emerson Jr. Jr and Woodring E Wright
    Citation: Skeletal Muscle 2011 1:12
  8. Sarcopenia, the loss of skeletal muscle mass during aging, increases the risk for falls and dependency. Resistance exercise (RE) training is an effective treatment to improve muscle mass and strength in older ...

    Authors: Christopher S Fry, Micah J Drummond, Erin L Glynn, Jared M Dickinson, David M Gundermann, Kyle L Timmerman, Dillon K Walker, Shaheen Dhanani, Elena Volpi and Blake B Rasmussen
    Citation: Skeletal Muscle 2011 1:11
  9. The most remarkable feature of skeletal muscle is the capacity to adapt its morphological, biochemical and molecular properties in response to several factors. Nonetheless, under pathological conditions, skele...

    Authors: Gabriella Dobrowolny, Michela Aucello and Antonio Musarò
    Citation: Skeletal Muscle 2011 1:3
  10. Skeletal muscle wasting is a major component of cachectic states found in a variety of disease settings, including cancer. As increasing caloric intake often provides little benefit in combating muscle loss in...

    Authors: Se-Jin Lee and David J Glass
    Citation: Skeletal Muscle 2011 1:2

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